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该患者是一名来自中国的50岁男子，他于1月8日至12日访问了武汉。 1月14日，他出现了干咳和轻微的畏寒感，所以这是第一天）。但是，他最初并没有寻求医疗救助，继续工作直到1月21日。他于1月21日去了一家医疗诊所，因为那时他已经出现了恶化的症状。他发烧，发冷，疲倦，咳嗽和呼吸急促。
麦克汉森(Mike Hansen)博士在此视频中分析了冠状病毒（COVID-19）尸检报告。
冠状病毒或它的新名称SARS-CoV-2是导致Covid-19爆发的病毒。中国武汉是这种流行病的中心，但是像安东尼·富奇博士这样的专家现在却说我们正处于大流行的边缘。
在获得Covid-19病人的尸检结果之前，了解被感染的总人数，冠状病毒性肺炎的总人数，罹患ARDS的人数以及死亡总数的背景非常重要。
查看这些数字时，我们应该意识到，在中国几乎可以肯定它们的漏报率是有原因的，其中有很多原因，我现在不再赘述。
尽管这些不是具体数字，但在这一点上我们必须经过。人数百分比。
同样，由于缺乏尸检和活检结果，到目前为止，尚无关于该病的病理报告。
但在2月17日发表于Lancet Respir Med的一项新的病例报告研究中，为死于Covid-19的患者提供了尸检结果。
急性COVID-19的病理发现
呼吸窘迫综合征
该患者是一名来自中国的50岁男子，他于1月8日至12日访问了武汉。 1月14日，他出现了干咳和轻微的畏寒感，所以这是第一天
的疾病）。但是，他最初并没有寻求医疗救助，直到1月21日才继续工作。他于1月21日去了一家医疗诊所，因为那时他已经出现了恶化的症状。他发烧，发冷，疲倦，咳嗽和呼吸急促。
这是他的胸部X光片。
1月22日（疾病发生的第9天），北京市疾病预防控制中心（CDC）通过反向实时PCR检测证实该患者患有COVID-19。
他立即被送进隔离病房，并通过口罩接受了补充氧气。
图片：这不是真正的病人，但我想给您一个关于我在这里谈论的内容的图像。
他给了他几种不同的药物，包括吸入型干扰素α-2b，洛匹那韦加利托那韦作为抗病毒治疗，以及
莫西沙星，以防止继发细菌感染。
还给他服用了类固醇甲基强的松龙，以减轻肺部炎症。
在发病的第12天，初次就诊后，除了发烧外，他的症状没有改善，他为此接受了药物治疗。
他在第12天的胸部X光片显示进行性双侧浸润。他一再拒绝在重症监护病房使用呼吸机，这显然是因为他患有幽闭恐惧症。
他的血氧饱和度值降低到60％，并且患者出现了心脏骤停。那时，他被插管了机械通气，胸部受压，肾上腺素。
不幸的是，他们无法使他复活。
进行尸检，并从肺，肝和心脏取活检样本。
心脏组织基本正常。
该患者的肝活检显示中度微血管脂肪变性和
轻度的小叶和门脉活动，表明该损伤可能是由SARS-CoV-2感染引起的，也可能是药物引起的肝损伤。
现在，进行肺活检。
肺组织的组织学检查显示，弥漫性肺泡损伤伴有细胞纤维粘液样渗出物，以及肺细胞的脱落和透明膜的形成。
这些发现与急性呼吸窘迫综合征一致。
在两个肺中均可见到以淋巴细胞为主的间质单核炎性浸润。有具有非典型大肺泡的多核合胞细胞，其特征在于突出的核仁，与病毒细胞病变样变化一致。
COVID-19的这些病理特征与SARS和中东呼吸综合征（MERS）冠状病毒感染中所见非常相似。
Coronavirus (COVID-19) Autopsy Report is analyzed in this video by Dr. Mike Hansen.
Coronavirus or more appropriately its new name SARS-CoV-2 is the virus responsible for the Covid-19 outbreak. Wuhan, China has been the epicenter of this epidemic, but some experts, like Dr. Anthony Fauci, are now saying that we are on the verge of a pandemic.
Before I get to the autopsy results of a patient with Covid-19, its important to understand the context of the numbers of total people infected, total people with coronavirus pneumonia, number of people who developed ARDS, and the total number of deaths.
When looking at the numbers, we should realize that they are almost certainly being underreported in China, and there are multiple reasons for that, which I won't get into right now.
Although these are not concrete numbers, its what we have to go by at this point. The percentage of people.
Also, up to this point, there has not been any pathology reported on this disease because of limited access to autopsy and biopsy results.
But finally, we now have a new case report study in Lancet Respir Med, published Feb 17, that has autopsy results for a patient who died from Covid-19.
Pathological findings of COVID-19 associated with acute
respiratory distress syndrome
The patient is a 50-year-old man from China, who visited Wuhan Jan 8–12. On Jan 14, he developed a dry cough and some mild chills, so this is day 1
of illness). However, he did not initially seek medical attention and kept working until Jan 21. He then went to a medical clinic on Jan 21, because by that time, he had developed worsening symptoms. He had fever, chills, fatigue, cough, and shortness of breath.
Here is his Chest x-ray.
On Jan 22 (day 9 of illness), the Beijing Centers for Disease Control (CDC) confirmed by reverse real-time PCR assay that the patient had COVID-19.
He was immediately admitted to the isolation ward and received supplemental oxygen through a face mask.
Picture: This is not the actual patient, but I wanted to give you a visual of what I’m talking about here.
He was given several different medications, which included the inhaled version of interferon alfa-2b, lopinavir plus ritonavir as antiviral therapy, and
Moxifloxacin, to prevent secondary bacterial infection.
He was also given a steroid, methylprednisolone, to attenuate lung inflammation.
On day 12 of illness, after the initial presentation, his symptoms did not improve, other than his fever, which he received medication for.
His chest x-ray on day 12 showed progressive bilateral infiltrates. He repeatedly refused ventilator support in the intensive care unit repeatedly, apparently because he suffered from claustrophobia.
His oxygen saturation values decreased to 60%, and the patient had a cardiac arrest. At that point he was intubated with mechanical ventilation, he had chest compressions and epinephrine.
Unfortunately, they are unable to revive him.
An autopsy is done, and biopsy samples were taken from the lung, liver, and heart.
The heart tissue was essentially normal.
The liver biopsy of this patient showed moderate microvascular steatosis and
mild lobular and portal activity, indicating the injury could have been caused by either SARS-CoV-2 infection or as a result drug-induced liver injury.
And now, to the lung biopsy.
Histological examination of lung tissue showed diffuse alveolar damage with cellular fibromyxoid exudates, along with the desquamation of pneumocytes and hyaline membrane formation.
These findings are consistent with acute respiratory distress syndrome.
Interstitial mononuclear inflammatory infiltrates, dominated by lymphocytes, was seen in both lungs. There were multinucleated syncytial cells with atypical large alveoli characterized with prominent nucleoli, consistent with viral cytopathic-like changes.
These pathological features of COVID-19 greatly resemble those seen in SARS and Middle Eastern respiratory syndrome (MERS) coronavirus infection.
评论：
The most disturbing thing about his autopsy is he had early steroids.
关于他的尸检，最令人不安的是他在早期服用类固醇激素。
Agreed. Allopathic medicine is retarded.
Can you give a explanation for the people that just drop dead while walking and some seem to have seizures or sever shaking ?Ive seen a lot of leaked videos of this from Wuhan. Anyway thanks for the video
您能为那些在步行中跌倒而有些似乎癫痫发作或剧烈颤抖猝死的人做一个解释吗？我从武汉看过很多泄露的视频。无论如何，谢谢你的视频。
It's a cytokine storm
这是细胞因子风暴
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俄国新冠病毒定义令人震惊 黑龙江省疫情特殊原因何在?
十分精彩独到的深刻分析。
对于极端看重俄国的习近平来说，能否控制住黑龙江的疫情具有特殊的意义。
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美丽冻人？回家乡村口玩雪吃雪的黑龙江兴凯湖女孩
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乌克兰群众对武汉撤回者如此这般
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郭文贵2月22日 ： 瘟疫中国政治派系和家族目前动向－－朱镕基全家撤回纽约 盛赞“大－纪－－元－－时==报”
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美国媒体最新访谈台湾最健壮网红“馆长”－－中国的伎俩 自由世界的反击还没有真正开始
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川普刚刚在拉斯维加斯的群众集会上激情四射，势不可挡，天地神人无一不服之，中国的未来布局已尽在掌握之中